Citrobacter freundii are facultative anaerobic Gram-negative coliform bacteria in the Enterobacteriaceae family. The bacteria are long rod-shaped with a typical length of 1-5 μm. Most C. freundii cells are surrounded by several flagella used for locomotion, but a few are non-motile.

The Citrobacter species, including Citrobacter freundii, are aerobic gram-negative bacilli. Citrobacter freundii are long rod-shaped bacteria typically 1-5 μm in length [1]. Most C. freundii cells are surrounded by many flagella used to move about, but a few are non-motile.

Members of the genus Citrobacter are gram-negative, non-sporing rods belonging to family Enterobacteriaceae and, as the name suggests, usually utilize citrate as a sole carbon source. The genus now consists of 11 genomospecies separable by their biochemical characteristics.

Bacteria Collection: Citrobacter freundii Additional Information

MODE OF TRANSMISSION: Citrobacter may be spread by direct contact with hospital staff members, mother to child transmission or through ingestion of environmental sources (fecal-oral route) but person-to-person transmission is more prevalent 7, 11.

Objectives: Citrobacter freundii is a frequent cause of nosocomial infections and a known cause of diarrheal infections, and has increasingly become multidrug resistant (MDR).

Various types of antibiotics, including amino- glycosides carbapenems, cephalosporins, chloramphenicol and quinolones, are used for the treatment of C. koseri infections. Treatment options can be monotherapy, with agents potentially active against Citrobacter spp. or combination therapy [8].

The strain was identified as C. freundii using Vitek 2 compact system and confirmed by 16S rDNA sequencing. CWH001 was resistant to nearly all tested antibiotics including aminoglycosides, cephalosporins, carbapenems, fluoroquinolones and sulfonamides, but remained susceptible to tetracycline (Table 1).

In patient with Citrobacter infections, the bacteria can be transmitted vertically from mother or horizontally from carriers or other hospital sources (14). The infection may occur as sporadic cases or nosocomial outbreaks.

Citrobacter freundii and Citrobacter koseri can cause urinary tract infections, and are found in wound, respiratory, meningitis, and sepsis. They can cause healthcare-associated infections, especially in pediatric and immunocompromised patients [41].

Citrobacter freundii is a species of facultative anaerobic gram-negative bacteria of the family Enterobacteriaceae. The bacteria have a long rod shape with a typical length of 1–5 μm. Most C. freundii cells generally have several flagella used for locomotion, but some do not and are non-motile.

Citrobacter isolates are reported to be the third most common organisms causing UTI in hospitalized patients after Escherichia More Details coli and Klebsiella species accounting for 9.4% of all isolates.

This is because UTIs are not sexually transmitted, and one partner does not spread the bacteria to the other. Instead, sex increases the risk of UTIs by introducing bacteria into the urethra. Any genital contact can introduce bacteria into the urethra, with or without a condom or penetration.

Treatment is with an aminoglycoside or an extended-spectrum cephalosporin. Almost all isolates are ampicillin resistant. The fatality rate for Citrobacter infections in newborns and older immunocompromised patients with Citrobacter pneumonia has been said to be high.

[1,2] The genus Citrobacter is distinct group of aerobic, gram negative bacilli from the Enterobacteriaceae family, widely distributed in water, soil, food and intestinal tract of man and animals. UTIs caused by Citrobacter species have been described in 5 to 12% of bacterial urine isolates in adults.

It is unlikely for anyone to get a UTI or STD from a toilet seat, as the urethra in males and females typically wouldn’t touch the toilet seat.

koseri is the only citrobacter to be commonly indole-positive), ferment lactose (C. koseri is a lactose fermentor), and use malonate. Citrobacter shows the ability to accumulate uranium by building phosphate complexes….

koseri mandates antimicrobial therapy based on the sensitivity of the pathogen microorganism. Various types of antibiotics, including aminoglycosides carbapenems, cephalosporins, chloramphenicol and quinolones, are used for the treatment of C. koseri infections.

Citrobacter koseri, formerly known as Citrobacter diversus, is a gram-negative bacillus that causes mostly meningitis and brain abscess in neonates and infants.

Patients with respiratory Enterobacter cloacae suffer from shortness of breath, yellow sputum (phlegm), fevers and heavy coughing. Interestingly, pneumonia caused by this bacterium often makes patients feel less ill than pneumonia caused by other bacteria, but has a surprisingly high mortality rate.

Enterobacter species, particularly Enterobacter cloacae, are important nosocomial pathogens responsible for various infections, including bacteremia, lower respiratory tract infections, skin and soft-tissue infections, urinary tract infections (UTIs), endocarditis, intra-abdominal infections, septic arthritis.

Enterobacter cloacae (urinary tract infections, respiratory tract infections) Escherichia coli (food poisoning, enteritis)

Yes there is a treatment if you know what kind of organism it is. There are antibiotics and they are fairly effective against this kind of thing, but it depends on when you know what it is. But particularly enterobacter which is a so-called gram-negative bacteria, it can cause sepsis very rapidly.

The antimicrobials most commonly indicated in Enterobacter infections include carbapenems, fourth-generation cephalosporins, aminoglycosides, fluoroquinolones, and TMP-SMZ. Carbapenems continue to have the best activity against E cloacae, E aerogenes, and other Enterobacter species.

A class of broad-spectrum antibiotics called carbapenem may be used as a last resort to kill Enterobacteriaceae.

How is Enterobacter cloacae transmitted? Immunocompromised Patients are at risk if they come into direct or indirect contact with contaminated persons or objects. The pathogens can also be transmitted via contaminated infusion solutions or blood products.

Enterobacter cloacae is part of the normal flora of the gastrointestinal tract of 40 to 80% of people and is widely distributed in the environment (15, 19, 39).

Enterobacter cloacae is ubiquitous in terrestrial and aquatic environments (water, sewage, soil, and food). The species occurs as commensal microflora in the intestinal tracts of humans and animals and is also pathogens in plants and insects.

Enterobacter can be found on human skin, plants, soil, water, sewage, intestinal tracts of animals, including humans, dairy products; and clinical specimens such as feces, urine, blood, sputum, and wound exudates.